Weapons of Mass Destruction?
Up in the Air in the Bluegrass
by Campbell Wood
February 17, 2005
“Only an alert and knowledgeable
citizenry can compel the proper meshing of the huge industrial and military
machinery of defense with our peaceful methods and goals so that security
and liberty may prosper together.” President Dwight D. Eisenhower in his
1961 Farewell Address.
The Pentagon has proposed budget changes for 2006 which would delay the destruction
of 523 tons of chemical weapons stored at the Blue Grass Army Depot in Richmond,
about 30 miles outside of Lexington, and 2,611 tons at the Pueblo Army Depot
in Colorado.
This is contrary to Congress’s stated emphasis on accelerating the destruction
of these deteriorating weapons after September 11. The proposed delay, estimates
at 5 to 10 years, would cause the U.S. to fail to fulfill its commitment under
the international Chemical Weapons Convention, which sets 2012 as the deadline
for destruction of the weapons. The Pentagon is also considering transportation
of chemical weapons across state lines for incineration in distant states.
These revelations came from Pentagon internal documents acquired and
released by the Chemical Weapons Working Group (CWWG), based in Berea.
According to CWWG director, Craig Williams, “Kentucky and Colorado are being
used as ‘bill-payers’ for huge cost overruns at the other disposal facilities,
particularly those using incineration. In addition, with neutralization demonstrating
it’s superior destruction and safety capabilities there’s the real possibility
that incineration advocates want to see this technology killed, as it would
inhibit their ability to push incineration on the world market.”
“No process is perfect,” adds Williams. “The fundamental difference between
the two approaches is the level of control operators have over the material
during processing. Neutralization is a low temperature, low air velocity process
which is very controllable. Incineration is high temperature, high air velocity
process with an exhaust stack at the other end. These incinerators have a
history of releasing agent and chronically emitting heavy metals, dioxins,
furans and other carcinogens. Neutralization eliminates such health threats.”
A document dated December 21, 2004, an Acquisition Decision Memorandum,
signed by Department of Defense Acquisition, Technology & Logistics Under
Secretary Michael Wynne, questions the design readiness of neutralization
for the disposal of chemical weapons. Under the Assembled Chemical Weapons
Assessment (ACWA) program instituted by the Army in 1997 to study alternative
technologies for the destruction of chemical weapons, neutralization was
demonstrated and proven effective, accepted by scientists, endorsed by the
Army and certified by Congress. Neutralization has already been in use at
Aberdeen Proving Ground, Maryland, where the program to destroy chemical
warfare agents in bulk containers is near completion. A neutralization facility
at Newport Chemical Activity, Indiana, will begin operating soon. Strong
opposition to incineration of chemical weapons in Colorado and Kentucky brought
about the decisions to use neutralization followed by super critical water
oxygenation in Kentucky and biotreatment in Colorado. Operations and budgets
at the four chemical weapons incinerators in Oregon, Utah, Arkansas and Alabama
remain intact. These incinerators have been plagued with technical problems,
chemical releases, shut downs, and budget overruns.
Here's how the budget fiasco happened. Last year, based on the apparent
financial needs of all defense programs, the Pentagon formulated its Chemical
Weapons Disposal Program Cost Projections for fiscal year 2006, a budget
which totaled approximately $1.406 billion. A line item in the Defense
Budget is Chemical Agent and Munition Destruction, under which heading comes
the destruction of chemical weapons in Kentucky and Colorado. The funding
needs for the programs in Kentucky and Colorado were accounted for in that
original budget. $270 million dollars would be divided between the two states:
$135 million to each state for the design and construction of neutralization
facilities. In December of 2004 CWWG learned that the Pentagon had subtracted
$240 million from the Kentucky/Colorado funding and had reassigned it to other
chemical weapons destruction sites to cover ballooning expenses. This altered
budget was submitted to Congress on February 7. As it stands, the budget
provides only $30 million for Kentucky and Colorado to split. $15 million
is insufficient funding to proceed with the planned destruction of the weapons
stored at the Blue Grass Depot. "They are robbing our money to pay for poor
management, poor technology choices and skyrocketing costs at other sites,"
said Williams.
These severe funding cuts have halted construction and design in Colorado
, and Bechtel Parsons Blue Grass, the contractor handling the planned facility
construction in Richmond, stopped hiring and suspended all bids in early January
until matters are clarified. It took a 19 year battle carried out via public
forums, scientific research and intense politics for the Blue Grass to replace
the Army’s proposed incineration with neutralization. The decision to use
neutralization in Kentucky was made by the Army in 2003. Design has been
underway and construction was to begin soon.
“After the time and energy I have expended on ensuring these weapons are
disposed of in a safe and environmentally friendly manner, I am personally
insulted by the Department’s efforts to delay destruction and its suggestion
of transporting the weapons elsewhere,” said Senator Mitch McConnel in a lengthy
statement he issued.
Both Ben Chandler and Jim Bunning have sent letters to the Department of
Defense urging that no budget cuts or delays hinder the planned schedule to
destroy the chemical weapons in Richmond.
The January 16 Lexington Herald-Leader published a strong editorial decrying
the Pentagon’s proposed setback for the Blue Grass Army Depot. As a symbol
of protest the Richmond City Commission has passed an ordinance that
would make it a crime to transport chemical weapons within the city limits.
Seven of the eight states with chemical weapon storage sites have joined
to block interstate transport of the weapons. Senators and House Representatives
from both sides of the aisle in Colorado and Kentucky introduced a bill to
derail the Pentagon’s transportation study. Senators from Alabama, Oregon,
Maryland, Utah, and Indiana are also cosponsors. More senators from other
states may sign on.
Doug Hindman of Berea was one of the many residents of Madison County shocked
to learn in 1984 that the Army had stores of chemical weapons at the Blue
Grass Depot. It had been a well kept secret. He attended the public meeting
at which Army officials announced to a full house itls plans to ininerate
the weapons. From that meeting sprung the opposition to the incineration plan.
A retired EKU psychology professor, Hindman is now Chair of the Citizen’s
Advisory Commission (CAC), created by Congress in 1991 with its members appointed
by the governor. “It’s disappointing to have the funding pulled at this late
date,” said Hindman. “All these years the CAC has worked very hard to bring
about a consensus among the local citizens, the state and federal government
to eliminate these weapons of mass destruction as quickly and safely as possible.”
Continued storage of the chemical weapons in Richmond comes with increasing
risk. Williams said that a 2002 classified report to Congress by the Secretary
of the Army indicated Kentucky will be the chemical weapons stockpile site
at the highest risk for terrorism after 2007. The deterioration and instability
of the stored chemical weapons have long been a concern. Some of them have
been stored there for over 50 years. The weapons include 69,449 M-55
rockets containing GB (sarin) and VX nerve agents and 32,285 projectile rounds
loaded with GB, VX or HD (mustard). The almost seven-foot long M-55 rocket
contains 10 pounds of either VX or GB nerve agent. Each rocket also contains
M28 propellant, which degrades over time through a series of chemical reactions
that generate heat - heat that could build and cause autoignition. A
chemical stabilizer added to the propellent at the time of manufacture helps
control the chemical reactions and heat. However, the stabilizer itself is
subject to degradation, which means that eventually autoignition is possible.
Army analysis has concluded that the rockets are not in imminent danger of
autoignition. However, lightning strikes or earthquakes could cause autoignition.
Another problem is with leakage from aging munitions. The Depot has rigorous
security and a daily routine of monitoring the stored weapons. This is obviously
no picnic for the men and women who serve at the Depot. One can only wonder
about how the commanders and soldiers at the base feel about the proposed
delay (military personnel rarely go on the record with reservations about
policy).
That the Pentagon has proposed to study transportation of the weapons is
a surprising development, given that the Army had already reached conclusions
on the matter after exhaustive studies.
In a Record of Decision in 1988, the former Undersecretary of the Army,
James Ambrose, spelled out the reasons why the Army had ruled out transporting
chemical weapons in favor of onsite destruction. He noted that catastrophic
events are a constant concern at installations and the surrounding communities.
By keeping the destruction process at the sites of storage, that concern would
be absent from transportation corridors. “It is more important, in my judgment,”
stated Ambrose, “to note the vastly more complex security, emergency response,
and safety requirements associated with a transportation option and our much
greater ability to control those same requirements at fixed installations...Compared
to the Onsite Disposal alternative, any alternative that requires shipment
of agent and munitions to other sites for disposal would be accompanied by
handling risks, chance of transportation accidents, and threat of terrorist
activities, which in turn increase public health and safety risks.” In his
conclusion, Ambrose emphatically stated: “...no stockpile agents or munitions
will be transported to other storage installations or sites for destruction.
The Army will construct disposal facilities specific to each installation’s
inventory of lethal chemical agents and munitions.”
THE GULF WAR SYNDROME CONNECTION
In 1997 James J. Tuite, III, was Interdisciplinary Sciences Director for
the Chronic Illness Research Foundation. Prior to that he had been chief Senate
investigator into Gulf War Syndrome, to which task he brought a background
which included work for the Secret Service (duties included criminal investigation,
intelligence and foreign counter terrorism and technology R&D) and service
in the Vietnam War as an Army medical specialist.
At a CWWG conference Tuite read from a letter he had received in 1994 from
a sick Gulf War veteran.
“We had gas alarms go off several times,” Tuite read aloud. “We were
told they were all false alarms. We noticed what we thought were missiles
streaking across the sky. We were told these were shooting stars. We heard
loud explosions in the sky and saw bright flashes of light. We were told these
were sonic booms.”
“That veteran is now suffering from Gulf War syndrome,” Tuite said.
When Tuite began his investigation in 1994, the Pentagon was reporting zero
troops potentially exposed to chemical warfare agents during the Gulf War.
An array of disease symptoms exhibited by a growing number of troops was dismissed
as stress related. In 1996 the Pentagon acknowledged that as many as 500
troops may have been exposed to chemical weapons. In 1997 that number rose
to 100,000; then 134,000 in 2000. In 2002, the Pentagon acknowledged
as many as 140,000 troops may have been exposed to chemical warfare agents.
“We have documents that show that the Department of Defense (DOD)
purposely manipulated their data,” said Steve Robinson, Executive Director
of the National Gulf War Resources Center.
Robinson says an investigation by the General Accounting Office (GAO) found
the DOD studies flawed. Based on GAO studies, Robinson said that as many as
400,000 troops may have been exposed to chemical agents. In a report available
online the GAO states that the DOD’s “models were not fully developed for
analyzing long-range dispersion of CW agents as an environmental hazard.”
In 1997, using U.S. government satellite photos of the Persian Gulf area
during the 1st Gulf War along with meteorological data and apparent exposure
of U.S. troops to low levels of chemical agents, Tuite created a map of the
eight chemical storage sites in the U.S. The map shows circled territory
around each site that would be potentially endangered in the event of a catastrophic
event causing the release of chemical agent into the atmosphere. The danger
of exposure to any area would be contingent on many variables, such as the
nature of the incident, the quantity of agents released, and meteorological
conditions, such as wind direction, cloud cover and so on. The circle around
Richmond includes all of Kentucky and reaches into 10 other states.
“The very same individuals who misled Congress about the exposure of our
veterans to chemical warfare agents, and who are associated with withholding
vital information that delayed research into their exposures, are telling
U.S. civilians that incineration and low level exposure to many of the very
same compounds is safe,” said Tuite.
Gulf War Syndrome is a complex matter. Troops were exposed to an array of
toxins. These included chemical weapons agents dispersed by U.S. bombing of
Iraqi munitions and possibly other sites where chemical weapons had been deployed
by the Iraq military and possibly by direct chemical weapon attacks by Iraq
forces. Another exposure was to depleted uranium, a controversy being revisited
in the current occupation of Iraq. The air was polluted by burning
oil fields. Some troops may also have been adversely effected PB pills (pyridostigmnine
bromide), which were meant to protect GIs from one type of nerve gas in the
Iraq arsenal.
Sarin is the chemical warfare agent that has received the most attention
in the epidemiological investigations of Gulf War Syndrome.
During the initial bombing of Iraq during the first Gulf War, U.S., Czech,
French and British forces all had chemical weapons detection systems that
on numerous occasions went into alarm mode. A number of these alarms specified
the presence of sarin.
Early bombing in that campaign struck Khamisiyeh, an Iraq munitions storage
site. After the war, U.N. inspectors found evidence that chemical weapons
had been stored at Khamisiyeh, and they included munitions loaded with sarin.
Sarin does not readily burn, as do other chemical warfare agents. Tuite’s
studies, supported by U.S. Air Force weather reports and satellite imagery,
put forth the scenario that sarin had risen in a gaseous plume into the upper
atmosphere where weather conditions directed it right over where hundreds
of thousands of U.S. troops were stationed. Cloud cover and other meteorological
conditions held the sarin in that general area. Heavier than air, the sarin
precipitated from the atmosphere onto the troops.
The exposure would have ranged from low to lethal. At the time -- this would
be the period of January 17 through January 24 of 1991 -- many soldiers complained
of a range of symptoms, which became dubbed the “Saudi Flu.” Tuite’s basic
scenario became accepted, but various models applied to the events resulted
in different estimates regarding troop exposure.
In his reporting Tuite gave a listing of the common symptoms suffered by
those with GWS. Not all of the screened veterans had all of the symptoms,
but most had a majority of them, said Tuite. Those symptoms included “recurring
severe headaches; fatigue or weakness; joint and muscle pain; memory loss
or difficulty concentrating; recurring rashes; lumps at joint areas and under
the skin; depression; irritability; night sweats; insomnia; urinary urgency
and frequency; diarrhea, sometimes bloody, or constipation; gastrointestinal
disorders; dizziness or blackouts; blurry vision; photosensitivity; shortness
of breath; coughing; abnormal hair loss; bleeding gums; abnormal liver functions;
chest pains; sinus infections; and tremors.” There were also reported cases
of cancers, blood diseases and various other disorders.
Researchers found the symptoms of GWS to be the same as those suffered by
illness brought on by the sarin attacks in the subways of Tokyo in 1995. GWS
symptoms have also matched the ones of sickened pesticide workers. Jerry Buccafusco,
Professor of Pharmacology and Toxicology at Medical College of Georgia reported
that “many of the symptoms reported by agricultural workers who had been
subjected to repeated low-level intoxication are similar to those reported
by Gulf War Veterans.”
Of the 697,000 troops deployed to the Persian Gulf during the first Gulf
War, 342,265 have filed claims for disabilities. Of those claims 303,884 have
been processed, and 268,685 have been approved. The Veterans Administration
does not keep statistics on the nature of those claims. U.S. troops are not
alone in this. Soldiers from Australia, Canada, the Czech Republic, France,
Norway, and Great Britain have also come forward claiming similar illnesses.
Iraqis also had reported high rates of birth defects and numerous disorders
including of the nervous system. Tuite also reported that tens of thousands
of large mammals (camels, goats, and sheep), as well as birds and insects
were killed by the environmental conditions.
BACK TO THE BLUE GRASS
The Gulf War Syndrome shows the nature of the danger in our midst here in
the Blue Grass. The Blue Grass Army Depot needs to get rid of these weapons.
It’s not fair to prolong the situation which keeps the military personnel
at Blue Grass Army Depot exposed to the danger. The continued storage of the
weapons there pose a growing and far reaching, not a diminishing and locally
bound, danger to the communities of all creatures around the Depot.
Craig Williams has spent twenty years active in this matter. It’s been his
working life for the past 15 years.
He sees Mitch McConnell as a key figure at this point in history. As Senate
Majority Whip and as a long term member of the Senate Defense Appropriations
Committee, he’s vested with a good deal of clout. “Senator McConnell has promised
to use every measure at his disposal to ensure that the weapons stored in
Kentucky are destroyed in a timely manner,” Williams said. “He has stated
his determination to resolve this funding crisis.”
The Herald-Leader reported earlier this week the fact that the Pentagon
has “on hand enough money to keep work going for the next couple years,”
but that it’s been lost amid the controversy--while “the Pentagon has impounded
more than $400 million” in funds appropriated by Congress, the decision-makers
for military spending, for neutralization plants in nearby Madison County,
and in Colorado.
Sidebar:
Bio Chem 101
Sarin belongs to the family of toxic nerve agents known as organaphosphates.
VX, also stored at the Blue Grass Depot, is an organaphosphate. Organaphosphates
are widely used as insecticides and pesticides. The soldiers in the first
Gulf War also doused themselves with organaphosphate insecticide to ward off
the desert fleas. The effects of low level exposure to sarin and the effects
of multiple low level toxic exposures over time became a subject of scientific
inquiry. Scientists concluded that repeated low level exposures to sarin
and/or other organaphosphates could lead to diseased states. This of course
highlights a concern for the agricultural communities that surround some
of the chemical weapons stockpiles in the U.S.
After years of studying Gulf War Syndrome, epidemiologist Dr. Robert Haley
at the University of Texas Southwestern Medical Center in Dallas established
that a significant number of Gulf War veterans had returned with something
that was more than stress induced: brain damage. He concluded that exposure
to sarin was a chief cause. Haley’s research has also revealed why some GIs
became ill and others didn’t, and the reason is genetic. Some GIs were gifted
with a gene that produces a certain enzyme, type Q paraoxonase, which fights
off sarin. Actually the enzyme is very specific in combatting sarin, the nerve
agent soman, as well as the common pesticide diazinon. For those that lacked
the protective enzyme, sarin damaged deep structures in the brain damaging
the parasympathetic nervous system, which Haley describes as the controller
of all ongoing unconscious functions in the body.
The National Medical Institute under the national Academy of Sciences concluded
that brain damage or neurological disease can be caused by exposures to low
level or non-lethal amounts of sarin, and the symptoms may emerge sometime
after initial exposures. The delayed toxic effects of chemical warfare agents
had been documented well before the Gulf War.
James J.Tuite,III, was Interdisciplinary Sciences Director for the Chronic
Illness Research Foundation stated, “...other nations, including Iraq and
the former Soviet states, acknowledge that repeated exposure to even very
low levels of chemical warfare agents can result in serious physiological
damage.”
Another scientist who studied Gulf War Syndrome was Howard B. Urnovitz,
Ph.D, who got his doctorate in microbiology and immunology at the University
of Michigan. Urnovitz found that chronic diseases are caused not by germs,
but by “junk DNA,” those strands of DNA that have been regarded as useless
excesses of DNA material. “What we learned by simultaneously studying GWS,
cancer, AIDS and multiple sclerosis is that the genes have the ability to
‘reshuffle’ and create new genes. We reason that these new genes are used
to adapt to the toxic environment in which we live. It seems that there are
confounding events that turns this reshuffling mechanism from a normal protective
process to a disease state.” Urnovitz suggests that like the mapping of the
normal human genome, a mapping of the “detours” taken by the human genome
under toxic exposure and chronic disease can be accomplished. He sees such
mapping as a potentially useful tool for medical diagnosis and treatment.